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Ester C (Vitamin C)

Ascorbic acid

In most animals, the water soluble nutrient, ascorbic acid, is synthesised from glucose. In humans a genetic lesion prevents us from making it and diet is the sole source of this vitamin. As a cofactor for many enzyme systems, ascorbic acid is involved in the synthesis of collagen, carnitine, endocrine hormones, and neurotransmitters. Many of the symptoms of the vitamin C deficiency disease scurvy can be attributed to weakened collagen structures, including bruising, muscle weakness, gum deterioration, and poor wound healing.

ESTER-C

The body changes vitamin C into numerous metabolites that may have physiological actions that are different from those of vitamin C itself. And these metabolites - whether manufactured in the body or ingested along with supplemental vitamin C - may influence how the vitamin itself is transported and utilized. The level of these metabolites in the body may not rise significantly, however, until very large doses of vitamin C are ingested. Laboratory studies have shown that suggested the mechanism by which C metabolites, especially threonate, might cause increased blood levels of vitamin C. Dr. Anthony Verlangieri at the University of Mississippi used isolated cells in culture to model how vitamin C is utilized by various body tissues. They observed that exposing cells to calcium threonate increased the uptake of ascorbic acid by these cells (Fay & Verlangieri, 1991; Fay, 1992; Fay, Bush & Verlangieri, 1994).